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About Engqvist A, Colmsjö A, Skare I.

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So far Engqvist A, Colmsjö A, Skare I. has created 991 blog entries.

Speciation of mercury excreted in feces from individuals with amalgam fillings.

Investigators established methods for the analysis of total mercury (Hg-total), oxidized mercury and mercury bound to sulfhydryl groups (Hg-S), mercury vapor (Hg0), and mercury from amalgam particles (APs) in fecal samples. Two individuals consumed mercury as a mercury-cysteine complex mercury vapor, and mercury from amalgam particles, and the cumulative excretion of mercury in feces was followed. Investigators found that 80% of the mercury from amalgam particles and mercury bound to sulfhydryl groups was excreted, but only 40% of the mercury vapor was excreted. Speciation of mercury excreted in feces from 6 individuals with a moderate loading of amalgam fillings showed that most of the mercury originating from the fillings consisted of oxidized mercury, which was probably bound to sulfhydryl-containing compounds. The proportion of amalgam particles in fecal samples from these individuals was low, and it did not exceed 26% of the total amount of mercury excreted.

By |2018-07-20T17:59:35+00:00January 1st, 1998|Mercury|

Observations on health before and after amalgam removal.

From 1972-1993 (5 years internship and 16 years of my own practice) amalgam was by far the most prevalent dental filling material used by me (in the side tooth area). Different reasons led me to abandon use of this material in the spring of 1994. An amalgam-critical work which had appeared in the FASEB journal in 1989 (Hahn et al.) was certainly an important reason for my decision. Moreover, some of my own earlier observations (e.g. with Pat. No 2 and Pat. No 73) and the fact that alternatives to amalgam were widely available, were equally important reasons for me to discontinue using amalgam, a material I had considered highly reliable before.

By |2018-07-20T17:48:32+00:00January 1st, 1998|Mercury|

Neuralgia-inducing cavitational osteonecrosis.

Unfortunately, the study presented was not well designed, consisting as it did of disparate clinical findings and uncontrolled treatment attempts that fail to illuminate the true nature of a controversial condition. In addition, the implied recommendations for treatment include medications that are far from innocuous and have the potential for serious side effects. Although the authors offer their own evaluation of this study, there are several problems with this work that they do not identify,

By |2018-08-25T17:14:28+00:00January 1st, 1998|Periodontal Disease|

Mercury in dental amalgam: a risk analysis.

With present knowledge it is impossible to estimate the risk of effects on the foetal brain induced by the mother’s exposure to mercury from amalgam. Available facts, however, do not support a dismissal of the risk. Therefore treatment of children and women of childbearing age with amalgam should be avoided. It is also recommended that use of amalgam for dental restorations in the population in general is abandoned and substituted with less toxic material, whenever this is available and affordable.

By |2018-07-18T23:24:10+00:00January 1st, 1997|Mercury|

Evidence that Mercury From Silver Dental Fillings May Be an Etiological Factor in Reduced Nerve Conduction Velocity in Multiple Sclerosis.

Seven multiple sclerosis subjects had their silver dental fillings (amalgams) removed which contained approximately 50% mercury. A visual evoked response (VER) test was performed before amalgam removal. Approximately six months after amalgam removal a second VER test was performed on all subjects, and the latencies of the VER decreased significantly. The mean latency of P1 for the right and left eye combined decreased by 23.1 milliseconds (P = 0.011) and N1 for the right and left eye combined decreased by 23.1 milliseconds (P = 0.026). It was hypothesized that mercury from dental amalgam was an etiological factor in reduced nerve conduction velocity in MS subjects.

By |2020-04-07T20:03:35+00:00January 1st, 1997|Mercury|

Evidence that mercury from dental amalgam may cause hearing loss in multiple sclerosis patients.

Seven female subjects diagnosed with mulitple sclerosis were tested for hearing at threshhold frequencies of 250, 500, 1000, 4000 and 8000 Hz. The subjects then had their silver dental fillings (amalgams) removed. Between six and eight months after amalgam removal, testing for hearing was repeated. Six of the seven subjects showed improvement in hearing of the right ear and five of the seven showed improvement in the left ear. Four of the six frequencies tested in the right ear improved significantly and three of six improved significantly in the left ear. The total frequencies were averaged before amalgam removal and compared to after amalgam removal. Hearing improved an average of 8 db (p=0.02).

By |2020-03-31T22:19:23+00:00January 1st, 1997|Mercury|

Mercury levels in plasma and urine after removal of all amalgam restorations: the effect of using rubber dams.

RESULTS:
After removal of all amalgam restorations, only the non-rubber dam group showed significant increases in the mercury levels found in plasma (p = 0.012) and urine (p = 0.037). However, one year later, the mercury levels in plasma and urine had sunk significantly below the pre-removal levels for both groups. When the changes in the mercury levels found were compared between the groups, the non-rubber dam group showed a significantly higher increase of mercury in plasma than the rubber dam group the day after removal (p = 0.0010). Compared to the pre-removal mercury levels in plasma and urine, the levels found 1 y after removal of all amalgam restorations were on average 52 +/- 23% (range 4-89%) lower in plasma and 76 +/- 21% (range 20-94%) lower in urine.

SIGNIFICANCE:
The study showed that dental amalgam had a statistically significant impact on the mercury levels found in plasma and urine in the patients tested, and that the use of a rubber dam during removal of all amalgam restorations significantly reduced the peak of mercury in plasma following removal.

By |2018-07-18T21:52:58+00:00January 1st, 1997|Mercury|

Alveolar osteonecrosis

We appreciate being given an opportunity to respond to the concerns expressed by Dr. Marilyn Moffat, President of the American Physical Therapy Association. Dr. Moffat takes us to task for having conducted a strict review of the literature in which we included only those studies that used control groups. We were surprised at Dr. Moffat’s comments, as our mandate was to present “the results from rigorous and methodologically sound clinical trials and studies published in peer reviewed journals.” This was explained very clearly by James Lipton in the introduction to this series of articles,
which were part of the Health Technology Assessment Conference on Management of Temporomandibular Disorders (TMD), sponsored by the National Institute of Dental Research and the National Institutes of Health Office of Medical Applications of Research.

By |2018-08-25T00:19:19+00:00January 1st, 1997|Other|

On the instability of amalgams.

In this web-document by Ulf Bengtsson you will learn more about the background, and you will find a description of an experiment performed in 1986 with thorough pictorial documentation, including a QuickTime movie showing how the mercury droplets form on the surface of non-gamma-two amalgams. All fully referenced. When you look at these droplets you actually look at something the establishment of the dental and medical communities don’t want to see, and also have gone into considerable efforts not to be made seen by others. What you see is a real phenomenon.

By |2018-07-18T21:21:26+00:00January 1st, 1997|Mercury|

Symptoms and differential diagnosis of patients fearing mercury toxicity from amalgam fillings.

Clinical signs, somatic symptoms reported by patients, and mercury excretion in urine were studied for 348 patients selected by odontologists or internists as amalgam-free referents, or as subjects with unexplained clinical findings or who were self-selected due to their fear of mercury intoxication from their amalgam fillings. Sixty patients were excluded because other explanations could be given for their complaints. The age distribution was bimodal, with peaks between 30 and 35 years and between 45 and 50 years. Mercury was determined in a morning urine sample and 30 minutes after the injection of 300 mg of 2,3 dimercapto-1-propane sulfonic acid (DMPS), a mercury-chelating agent. The patients were followed for 1-3 years. Among the patients there were 26 who had had their amalgam fillings removed and who, at the time of the follow-up, were subjectively cured. When the patients were classified according to the excretion of mercury after the DMPS challenge, those who belonged to the upper quartile had an odds ratio of 7.2 (95% confidence interval 3.1-15.2) for becoming cured after amalgam removal. The symptoms of the cured patients had been predominantly mental. No consistent clinical picture could, however, be found among the other patients, as various types of mental and physical distress were reported.

By |2018-07-31T21:12:44+00:00January 1st, 1997|Mercury|
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