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Etiology and treatment of idiopathic trigeminal and atypical facial neuralgias.

In a series of sixteen patients with idiopathic trigeminal neuralgia and twenty-one patients with atypical facial neuralgia, it was found that the painful phenomena associated with both disorders were, in nearly all instances, closely related to the presence of maxillary or mandibular bone cavities at previous tooth extraction sites. Standard oral surgical procedures for curettage of the cavities, together with administration of antibiotics, were employed in the successful treatment of both the trigeminal and atypical facial neuralgias, with complete pain remissions for periods varying from 2 months (for most recently treated cases) up to 9 years. The observations and results of this study suggest that dental and oral disorders may play a role in the genesis of trigeminal and atypical facial neuralgia.

By Roberts AM, Person P.|2018-08-29T21:40:11+00:00January 1st, 1979|Other|

Jawbone cavities and trigeminal and atypical facial neuralgias.

The possible role of dental and oral disease in the etiology of idiopathic trigeminal and atypical facial neuralgias has been examined. Among thirty-eight patients with idiopathic trigeminal neuralgia and twenty-three patients with atypical facial neuralgia, there was in nearly all instances a close relationship between pain experienced and the existence of cavities in alveolar bone and jawbone of the patients. The cavities were at the sites of previous tooth extractions and, although at times more than 1 cm. in a given diameter, were usually not detectable by x-rays. A new method for their detection and localization was developed empirically, based on the observation that peripheral infiltration of local anesthetic into or very close to the bone cavity rapidly abolished trigger and pain perception by patients during persistence of the anesthetic action. Histopathologic examination of bone removed from cavities by curettage revealed, in both idiopathic trigeminal and atypical facial neuralgias, a similar pattern characterized by a highly vascular abnormal healing response of bone. Some lesions presented a mild chronic inflammatory (lymphocytic) infiltration. Preliminary microbiologic studies of material from the walls of the cavities showed the existence within them of a complex, mixed polymicrobial aerobic and anaerobic flora. Treatment consisted of vigorous curettage of the bone cavities, repeated if necessary, plus administration of antibiotics to induce healing and filling-in of the cavities by new bone. Responses of patients to the above treatment consisted of marked to complete pain remissions, the longest of which has been for 9 years. Complete healing leads to complete and persistent pain remissions. It was concluded that in both idiopathic trigeminal and atypical facial neuralgias, dental and oral pathoses may be major etiologic factors.

By Ratner EJ, Person P, Kleinman DJ, Shklar G, Socransky SS.|2018-08-28T21:17:55+00:00January 1st, 1979|Periodontal Disease|

Zum Nachweis der Lyse von Hg aus Silber-Amalgam von Zahnfullun-gen.

As a dental donor, patients were selected who either because of necessary prosthetic restorations or
in children due to other conditions, which also made the removal of teeth necessary. In rare cases
pulpitis was the reason for extraction.

By Till T, Maly K.|2019-02-18T22:30:49+00:00January 1st, 1978|Other|

Osteomyelitis of the jaws.

Material consisting of 16 patients examined and/or treated under the diagnosis osteomyelitis is reported. A description is given of the history, etiology, clinical and radiographic findings, results of histologic and microbiologic examinations, blood chemistry, diagnostic subgroups, findings at operation and the surgical and antibiotic therapy used. The results of treatment at short-term follow-up are given. Of nine patients observed 5 months or longer, six showed signs of healing, while the result was judged as uncertain in two cases and unsuccessful in one.

By Rangne A, Ruud A.|2018-08-28T20:59:50+00:00January 1st, 1978|Periodontal Disease|

The effect of addition of some materials to saliva at different temperature degrees on electrogalvanism.

Electrogalvanism had been known at the end of the 18 th century. Since then, this subject has been discussed by many authors.po’) They found that when different metallic restorations are placed in the patient’s mouth electric current generated in the saliva. The intensity of this current depends on the electric potential difference of the metallic restorations,( 7) the fiuid film thickness( 4) and the surface area of the resto rations. (‘ ) Soft tissue irritation, (9) pain in and around the teeth,(lO) corrosion and discoloration of the metallic resotration( 4, 1 1) were considered to be the sequel of galvanism.

By Ramadan FA, Mohamed FA, Sabet EM|2018-08-28T20:47:30+00:00January 1st, 1978|Other|

Potential and polarization measurements in vivo of oral galvanism.

Galvanic currents within the oral cavity may have harmful effects on biological tissues. In the present work 16 patients with different kinds of oral and other discomfort and pain which they attributed to oral galvanism were investigated. The potential and polarization of each metal restoration within reach of a platinum probe were measured versus a reference electrode. A recording of these measured values permits a calculation of the currents which may pass between the teeth. A control group of patients with no subjective symptoms of galvanism in the oral cavity was also investigated. The results of the electrochemical measurements showed that conditions for oral galvanism existed within the individuals of the patient group as well as within the control group. One remarkable observation was that the metallic restorations often consisted of different electrically isolated areas with different electrochemical properties. This and other factors influencing oral galvanism are discussed.

By Bergman M, Ginstrup O, Nilner K.|2018-08-24T19:26:18+00:00January 1st, 1978|Mercury|

A clinical and laboratory study about the influence of temperature changes on electrogalvanism between similar and dissimilar, polished and unpolished metallic dental restorations.

Galvanism or galvanic action is a well known phenomenon in dentistry. Small galvanic currents are generated from the presence of dissimilar metals in most mouths and consequently cause corrosion of fillings.

By Younis MN|2018-09-01T18:51:43+00:00January 1st, 1978|Mercury|

Measurements of currents between dissimilar alloys in the oral cavity.

A method is described for the measurement of currents generated between dissimilar alloys in die oral cavity. Tests were performed using amalgam fillings versus specimens consisting of a gold alloy, a chromium-cobalt alloy and a stainless sleel alloy. Currents of 0.1-0.3I1Ncm2 of specimen area were measured.

By Gjerdet NR, Brune D.|2018-09-15T01:54:31+00:00January 1st, 1977|Mercury|

A galvanic study of different amalgams. Journal of Oral Rehabilitation.

Due to the difference in open circuit potential (OCP) versus SCE for Aristaloy amalgam (-969 mV) and Dispersalloy amalgam (-549 mV) in Ringer’s solution at 25 degrees C, a galvanic cell was created with Dispersalloy amalgam as cathode and Aristaloy amalgam as anode. The galvanic corrosion current was studied as a function of time for the above cell as well as for a cell of type III dental gold (OCP is +0-5 mV) versus Aristaloy amalgam. The initial corrosion current of the latter cell (105 micronA) is about twice that for the cell of Aristaloy amalgam versus Dispersalloy amalgam (54 micronA), however, their passivating behaviour is quite similar. Also, an interrupted galvanic corrosion test simulating the oral ‘make and break’ situation was performed. A much higher corrosion current than the steady state was found when the two electrodes resumed contact.

By Chen CW, Greener EH.|2018-08-25T02:19:01+00:00January 1st, 1977|Mercury|

Mercury intoxication in a dental surgery following unreported spillage.

An unreported spillage of mercury in a dental surgery resulting in four non-fatal cases of mercury intoxication by inhalation of mercury vapour is described and clinical symptoms in relation to urine mercury levels noted and discussed. The method of detection and decontamination of the mercury vapour source is reported, and suggestions for preventing spillage of mercury and for reducing the risk of intoxication from such accidental spillage are made. Continued vigilance by all persons handling mercury is stressed.

By Merfield DP, Taylor A, Gemmell DM, Parrish JA.|2018-07-26T16:57:32+00:00January 1st, 1976|Mercury|

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About Roberts AM, Person P.