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Environmental pollution by fluorides in Flathead National Forest and Glacier National Park.

Vegetation samples were collected in June and Oct. 1970 at varying distances from the Anaconda Aluminium Co. plant at Columbia Falls, Montana. Determinations were of foliar F concn., injury index II (an estimate of gross amount of visible injury or ‘burn’ for foliage of a given year), insect pest populations, and F concn. in insects collected within 0.5 mile of the Al reduction plant. Vegetation in control areas contained <10 p.p.m. F. II was shown to be a conservative parameter of pollution since many samples with high F concn. did not show injury. F concn. in vegetation samples was used as the basis of pollution mapping, whereby the pollution index in ‘isopols’ is equal to the local F conc. (with 10 as the background value). Visible injury occurred above 30 isopols (on a total area of 69 000 acres) in susceptible species, viz. pines (Pinus albicaulis, P. contorta, P. monticola and P. ponderosa) and Douglas fir (Pseudotsuga menziesii); the herb Smilacina stellata was a particularly sensitive indicator of F pollution. Nearly all vegetation was injured moderately at 100-300 isopols and (except grasses) severely at 300-600 isopols. Characteristics symptoms of F injury in conifers included enlarged cell nuclei and hypertrophy of resin-canal epithelium and vascular parenchyma.

By |2018-07-19T18:42:59+00:00January 1st, 1971|Fluoride|

Printing of mercury distribution on the surface of dental amalgams.

Mercury, an essential element of dental amalgams, is not a stable material but one that vaporizes at ordinary temperatures. By use of a simple technique, printings were obtained from mercury amalgam specimens but were not obtainable from alloy-zinc control specimens. Unburnished and unpolished specimens yielded more intense printings than burnished and polished specimens.

By |2018-08-13T18:42:00+00:00January 1st, 1970|Mercury|

The effect of fluorine on dental caries [editorial].

We do know the use of drinking water containing as little as 1.2 to 3.0 parts per million of fluorine will cause such developmental disturbances in bones as osteosclerosis, spondylosis, and osteopetrosis, as well as goiter, and we cannot afford to run the risk of producing such serious systemic disturbances in applying what is at present a doubtful procedure intended to prevent development of dental disfigurements among children.  […]  Because of our anxiety to find some therapeutic procedure that will promote mass prevention of caries, the seeming potentialities of fluorine appear speculatively attractive, but, in the light of our present knowledge or lack of knowledge of the chemistry of the subject, the potentialities for harm far outweigh those for good.

By |2018-07-23T20:57:43+00:00January 1st, 1944|Fluoride|

Chronic fluorine intoxication [editorial]. Journal of the American Medical Association.

Distribution of the element fluorine is so widespread throughout nature that a small intake of the element is practically unavoidable. Fluorides are general proto-plasmatic poisons, probably because of their capacity to modify the metabolism of cells by changing the permeability of the cell membrane and by inhibiting certain enzyme systems.

By |2018-07-23T21:10:06+00:00January 1st, 1943|Fluoride|

Hazard of mercury vapor in scientific laboratories.

This paper is divided into two parts. The first part reports the determination of the amounts of mercury vapor found in the air of various ventilated and unventilated laboratories at the National Bureau of Standards and elsewhere. The new optical mercury-vapor detector devised by Woodson and produced by the General Electric Co. was used to secure this information. The concentrations found ranged up to 70 micrograms of mercury per cubic meter of air. Various sources of mercury vapor are described, as well as some measures taken to lower the concentration.

By |2018-07-30T23:21:03+00:00January 1st, 1941|Mercury|

Mercury poisoning from the public health viewpoint.

MERCURY has ranked prominently among the causes of poisoning for several hundred years. It was known to the Egyptians in 1600 BC The knowledge of mercury accompanied the gradual spread of Egyptian culture to Greece, and from Greece the information passed to Rome, and thence over the Roman Empire. The Egyp-tians, Greeks, and Romans considered mercury too toxic for medicinal use. Mercury was known in India about 500 BC where its medicinal value was recognized during the 1st century AD, and mercury stomatitis was described during this time.

By |2018-07-26T23:06:47+00:00January 1st, 1938|Mercury|

Present Conceptions of Dental Focal Infection.

The purpose of this paper is to attempt to correlate the observations which have been made and the present conceptions of the relationship between dental infection and systemic disease. To these will be added the results we have obtained in attempting to apply the observations made by investigators in this field.

By |2019-06-03T19:51:49+00:00January 1st, 1936|Other|
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